The Mayday Project

06/01/2026

💚 LYME AWARENESS MONTH MAY BE ENDING… BUT THE SEARCH FOR TRUTH IS NOT. 💚

For more than a decade, The Mayday Project has dedicated thousands of hours to researching Lyme disease, tick-borne infections, emerging science, historical records, published studies, patents, and patient experiences that many believe have been overlooked for far too long.

While Lyme warriors have spent years fighting for recognition, science continues to uncover new complexities surrounding chronic infection, persistence, immune dysfunction, co-infections, and the long-term impact of tick-borne disease.

The Mayday Project has never stopped asking questions.

Never stopped researching.

Never stopped fighting for the people who were told:
“It’s stress.”
“It’s anxiety.”
“Your tests are normal.”

As Lyme Disease Awareness Month comes to a close, remember this:

Awareness was only the beginning.

Research continues.
Questions remain.
Voices are growing louder.

The Mayday Project remains committed to pursuing answers, sharing research, and pushing for greater transparency and investigation into Lyme disease and related illnesses.

Hope remains that the work of dedicated researchers, advocates, patients, and organizations will continue moving these conversations forward.

💚 For every warrior still suffering…
💚 For every family still searching…
💚 For every question still unanswered…

We keep going.

Because truth doesn’t fear investigation.

THE MAYDAY PROJECT
10+ Years of Research.
10+ Years of Advocacy.
10+ Years of Refusing to Give Up.

05/31/2026

Taxonomic fraud hides were biological warfare mutations connected to MK Naomi, Operation Chase, and Watergate. Watergate was really about the harmful effects of dumping biological, chemical, and nuclear materials into the environment. Presidents Lyndon Johnson and Richard Nixon were responsible for allowing this. JFK was killed because he tried to stop it, but the CIA and his Secretary of Defense wanted it to continue. Since the CIA controlled MK Naomi, this connects the CIA to JFK’s death. They say civilians have been used as test subjects for many years and claim science proves it.

To hide the damage done to living things, they claim scientists created a new kingdom of life, moved real bacteria into the Archaea group, and then said these organisms had just been discovered. These organisms were actually created and released into water. This is the real meaning behind “Watergate.”

The reclassification of living organisms by Woese and later changes to taxonomy rules were done to hide what they call the “Pseudomonadati kingdom” or a kingdom of “fake bacteria.” The classification rules accidentally exposed the problem.

The biology of these organisms supports their claim. Many organisms place in this group are called polyphyletic.

Polyphyletic means organisms are grouped together even though they came from different evolutionary origins. They may look similar or share certain traits, but they do not come from one common ancestor.

If a group is polyphyletic, it means the old classification system was artificial and did not reflect true relationships.

What people call “pseudoscience” is actually the study of thousands of biological warfare mutations. They argue that the term “pseudoscience” is used the same way as the term “conspiracy theorist” to discourage people from investigating these ideas. This was done to hide the damage caused by people trying to control or alter life itself.

05/27/2026

How does this apply to disease, specifically Borreliella burgdorferi?

For Borreliella burgdorferi B31, the combined molecular, structural, environmental, and taxonomic datasets support a ribosomal corruption model centered on a 70S prokaryotic ribosomal core of the tunicate Lokiarchaeum, operating alongside a severely fragmented viral-plasmid architecture in a constant state of flux. The organism retains a standard 70S ribosome for protein synthesis, but its major survival, persistence, immune evasion, and genomic transfer systems are distributed across linear plasmids, prophage-plasmids, and viral-style replicons rather than remaining confined to a said to be, single, stable bacterial chromosome.
The main linear chromosome itself displays viral-style structural organization through its covalently closed hairpin telomeres and ResT-mediated telomere resolution system. Linear chromosomes are found in viral systems and linear phage-plasmids, where hairpin telomeres protect exposed DNA ends from degradation. In Borreliella, the chromosome, lp25, lp28-1, and other linear replicons all rely on this same structural maintenance strategy. This places the chromosome within the same viral-style architectural framework as the organism's linear plasmid systems.
The viral chromosome serves as the core housekeeping platform, while lp25 provides metabolic persistence through the BBE22/pncA NAD+ salvage system, which is required for survival within mammalian hosts. lp28-1 functions as the immune-evasion system through the vls antigenic variation locus, where silent donor cassettes continuously rewrite the active vlsE expression site. The cp32 family serves as the prophage-plasmid layer that links the organism to viral recombination, genome transfer, prophage activation, and phage-associated restructuring. Together, these systems create a chimeric mosaic genomic structure in which essential survival traits are distributed across viral plasmid replicons, prophage systems, and linear (viral) chromosome architecture.
Under the broader definition of ribosomal corruption, the evidence chain links ancestral 70S ribosomal continuity, viral integration systems, lambdoid phage-associated transfer pathways, genome instability, recombination pressure, and taxonomic reassignment into a single interconnected framework. In this, Borreliella functions as a 70S-based, Pseudomonadati (fake units of bacteria) kingdom organism operating through layered viral-plasmid systems, prophage architecture, antigenic reshuffling, and fragmented extrachromosomal survival modules, producing a chimeric-mosaic presentation responsible for ongoing disease and disorder presentations that experience genetically shifting in response to environmental stress, thereby causing a chronic infectious state, additional taxonomic instability, and reclassification pressure with taxonomy, especially under the designation of Candidatus, meaning it genomic presentation is too fragmented to place it without a single genomic taxa. This is why the government lies about Lyme.

05/27/2026

What is a ribosome?
(simplified)
The combined molecular, structural, environmental, and historical evidence supports a model of ribosomal corruption centered on the ancient 70S ribosome and large-scale genome instability caused by environmental stress. Studies by James Lake showed that archaeal/eocytic ribosomes share important structural features with eukaryotic ribosomes. These include special ribosomal gaps, protrusions, and layered RNA expansion regions. At the same time, Lokiarchaeum and related Asgard systems contain eukaryotic-style proteins and structural scaffolding built onto a 70S-like ribosomal core. This creates a direct structural link between the ancestral 70S prokaryotic ribosome, archaeal systems, and the larger 80S eukaryotic ribosome discovered in the late 1970s.
Documented environmental exposure associated with Operation CHASE introduced chemical and radiological stress into marine environments. These stressors are capable of causing DNA fragmentation, double-strand breaks, phage activation, recombination, transduction, and genome integration events. Marine tunicates acted like biological concentration systems because they constantly filtered huge amounts of ocean water and trapped microorganisms, phages, symbionts, and damaged genetic material inside their bodies. This environment became directly associated with the Church Committee–documented release of deep-sea chemical, biological, and nuclear warfare waste connected to MKNAOMI-era activities and Operation CHASE. Because tunicates collected so much biological material in one place, damaged DNA, viral particles, symbionts, and mutagenic stressors were able to interact at unusually high levels among multiple organisms across multiple kingdoms.
Viral plasmids, prophages, tmRNA rescue systems, and lambdoid phage packaging pathways provide the biological mechanisms for cross-domain sequence transfer, genome instability, ribosomal corruption, and intracellular genetic alteration. Under intense stress conditions, phages can package damaged genetic material and transfer it between biological systems, thereby increasing recombination and genome instability. This creates chimeric-mosaic genomic patterns involving viral DNA, bacterial systems, archaeal structures, and eukaryotic-style ribosomal components. Today, the kingdom associated with the 70S rRNA is none other than the Pseudomonadati kingdom (Imperative taxonomy rule defining the taxa- Latin origin - fake units of bacteria).
Together, the evidence supports stress-driven mutation, recombination, ribosomal structural continuity, ribosomal corruption, and the formation of chimeric 70S/80S ribosomal structures linked to genome instability and taxonomic reassignment. The model connects the Lokiarchaeum tunicate symbiont system, the ancestral 70S rRNA ribosome, and the viral-layered 80S eukaryotic ribosome through post–Operation CHASE mutagenic stress fields across the land and sea, and genetically altered lambdoid phage activity. As these systems became more mixed and unstable, organisms no longer fit neatly into traditional classification systems, creating pressure for major taxonomic reassignment.

05/27/2026

What is a ribosome?
The combined molecular, structural, environmental, and historical datasets support a unified model of translation-system (ribosome) corruption centered on ancestral 70S ribosomal continuity and stress-driven genomic destabilization. High-resolution ribosomal studies by James Lake demonstrated structural continuity between eocytic/archaeal ribosomes and eukaryotic ribosomal architecture, while Lokiarchaeum and related Asgard systems contain eukaryotic-signature proteins and expansion-segment scaffolding layered onto a 70S-like core. Concurrently, documented environmental mutagenic exposure associated with Operation CHASE introduced intense chemical and radiological stress capable of driving DNA fragmentation, phage activation, transduction, recombination, and integration events. Viral integration systems, tmRNA-associated rescue mechanics, and lambdoid phage packaging pathways provide the mechanistic framework for genome instability, ribosomal corruption, and cross-domain sequence transfer by viral plasmids and prophages. Together, the evidence chain supports stress-induced mutation, recombination, ribosomal structural continuity, corrupt layering of translation systems, and the emergence of chimeric 70S/80S ribosomes linked to genomic instability and taxonomic reassignment. This evidence chain forms the direct connection between the Lokiarchaeum tunicate symbiont system, the ancestral 70S rRNA prokaryotic ribosome, and the viral-layered 80S rRNA eukaryotic ribosome architecture, with the post–Operation CHASE environmental stress field serving as the mutagenic trigger that links ribosomal structural transference to translation-system corruption, cross-domain genomic instability, intracellular corruption via endosymbionts (often incorrectly noted in literature as organelles), and subsequent taxonomic reassignment pressure due to chimeric-mosaic presentation involving genetic alteration associated with lambdoid phages.

05/13/2026

The revisionist history BS of biological, chemical, and nuclear warfare needs to stop already! Just because Richard Helm destroyed the files in 1973, in the middle of Nixon's Watergate investigation, doesn't mean the ramifications of the programs get to be swept under the rug.

05/13/2026

You’re not crazy; they just won’t admit to the damning truth to help you. They not only manipulated the organisms, but they then manipulated taxonomy to cover up their sins.

05/13/2026

Tell me what aspect of this organism, history, or taxonomy you would let to learn more about.

05/11/2026

It's important to understand the history and the reasons they hide the truth about Lyme and the many agents of horizontal gene transfer. This is the key to understanding what you suffer from.

05/10/2026